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Wikileaks: Form 3D2ABO220194403G (Republic of Korea FTA)
The South Korean government has agreed to lift its economical sanctions on American agricultural products and import additional meat products by the year 2015. President Lee Myung Bak has promised Obama that he will assure the South Korean citizens of the safety of the American bovine products by the end of his term as a president and import cows that are three years or older. President Lee also quoted that the South Korean citizens are becoming more trusting toward American beef products as time progresses. Many economists are optimistic in their views that if the Korean FTA approves of this trades agreement, United States will be able to get an extra boost in GDP, hopefully ending the economic crisis currently taking place. During cell division, enzymes that duplicate DNA cannot continue their duplication all the way to the end of chromosomes. If cells divided without telomeres, they would lose the ends of their chromosomes, and the necessary information they contain. The telomeres are disposable buffers blocking the ends of the chromosomes, are consumed during cell division, and are replenished by an enzyme, telomerase reverse transcriptase. In the early 1970s, Russian theorist Alexei Olovnikov first recognized the problem of how chromosomes could not completely replicate their ends. Building on this, and to accommodate Leonard Hayflick's idea of limited somatic cell division, Olovnikov suggested that DNA sequences are lost every time a cell/DNA replicates until the loss reaches a critical level, at which point cell division ends.[3][4] In 1975–1977, Elizabeth Blackburn, working as a postdoctoral fellow at Yale University with Joseph Gall, discovered the unusual nature of telomeres, with their simple repeated DNA sequences composing chromosome ends. Their work was published in 1978. The telomere shortening mechanism normally limits cells to a fixed number of divisions, and animal studies suggest that this is responsible for aging on the cellular level and sets a limit on lifespans. Telomeres protect a cell's chromosomes from fusing with each other or rearranging — abnormalities that can lead to cancer — and so cells are destroyed when their telomeres are consumed. Most cancers are the result of "immortal" cells that have ways of evading this programmed destruction.[5] Elizabeth Blackburn, Carol Greider, and Jack Szostak were awarded the 2009 Nobel Prize in Physiology or Medicine for the discovery of how chromosomes are protected by telomeres and the enzyme telomerase.[6] Telomeres are repetitive DNA sequences located at the termini of linear chromosomes of most eukaryotic organisms. Most prokaryotes, lacking this linear arrangement, do not have telomeres. Telomeres compensate for incomplete semi-conservative DNA replication at chromosomal ends. The protection against homologous recombination (HR) and non-homologous end joining (NHEJ) constitutes the essential “capping” role of telomeres that distinguishes them from DNA double-strand breaks (DSBs).[7]
